Advertisement
 

doctorslounge.com

 
Powered by
Careerbuilder

 

                    Home  |  Forums  |  Humor  |  Advertising  |  Contact
   Ask a Doctor

   News via RSS

   Newsletter

   Cardiology

   News

   Conferences

   CME

   Forum Archives

   Diseases

   Symptoms

   Labs

   Procedures

   Drugs

   Links
   Specialties

   Cardiology

   Dermatology

   Endocrinology

   Fertility

   Gastroenterology

   Gynecology

   Hematology

   Infections

   Nephrology

   Neurology

   Oncology

   Orthopedics

   Pediatrics

   Pharmacy

   Primary Care

   Psychiatry

   Pulmonology

   Rheumatology

   Surgery

   Urology

   Other Sections

   Membership

   Research Tools

   Medical Tutorials

   Medical Software

 

 Headlines:

 
 

Back to Cardiovascular Diseases

AV nodal reentrant tachycardia (AVNRT)

Atrioventricular (AV) nodal reentrant tachycardia is the most common type (excluding atrial fibrillation) of supraventricular tachycardia. It accounts for about 60% of all fast heart rates that start in the upper part of the heart (excluding atrial fibrillation).

AV nodal reentrant tachycardia (AVNRT) occurs in the presence of a reentrant circuit (an abnormal or extra electrical pathway in the heart, a kind of "short circuit.") involving two anatomically and physiologically distinct pathways (fast and slow pathways) linking the right atrium to the AV node. AVNRT is the most common paroxysmal supraventricular tachycardia (PSVT).

Symptoms and signs

These includes attacks of palpitations, dizziness and syncope, dyspnea, chest pain as well as anxiety. These attacks start and terminate abruptly (i.e. paroxysmal).

ECG readings

In typical (slow-fast) AVNRT, activation spreads from the atrium to the AV node via the slow pathway and returns to the atrium via the fast pathway. The ECG reveals a rate usually between l50-250 bpm-with a P wave obscured by the nearly synchronous atrial and ventricular depolarization-and a QRS complex that is either normal (base-line) or broadened, owing to rate-related aberrancy In atypical (fast-slow) AVNRT, the activation pathway is reversed, the rate is similar to that for typical AVNRT but an inverted P wave usually is readily apparent in the T wave, resulting in a normal or minimally prolonged PR interval.

Treatment

Initial therapy of acute episodes of narrow-complex tachycardias, particularly AVNRT includes vagal maneuvers (e.g. carotid massage, Valsalva maneuver) and, if unsuccessful, bolus administration of short-acting agents that slow or block AV nodal conduction, such as adenosine, verapamil, or diltiazem.

Chronic drug therapy may include calcium channel antagonists, beta-adrenergic antagonists, or digoxin. Radiofrequency catheter ablation now can obviate the need for such therapy in most patients.

advertisement.gif (61x7 -- 0 bytes)
 

Are you a doctor or a nurse?

Do you want to join the Doctors Lounge online medical community?

Participate in editorial activities (publish, peer review, edit) and give a helping hand to the largest online community of patients.

Click on the link below to see the requirements:

Doctors Lounge Membership Application


previous.gif (72x17 -- 347 bytes) next.gif (72x17 -- 277 bytes)
 

 advertisement.gif (61x7 -- 0 bytes)

 

 



We subscribe to the HONcode principles of the HON Foundation. Click to verify.
We subscribe to the HONcode principles. Verify here

Privacy Statement | Terms & Conditions | Editorial Board | About us
Copyright 2001-2012 DoctorsLounge. All rights reserved.